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Further experimentation focused on the effect of ASO treatment on specific striatal-enriched transcripts in six-month old YAC128 and control mice. These results showed that reducing mutant HTT expression using an ASO directed specifically against human being mutant HTT in YAC 128 mice considerably corrected the transcriptional profiles of DARPP32, enkephalin and CB1. D1 and D2 receptor levels showed a development towards improvement pursuing ASO treatment also, says Dr. Stanek. The authors claim that monitoring transcriptional adjustments could serve as a powerful tool for clinicians to follow HD progression and treatment. Since taking samples from human brain isn’t possible, alternative methods, such as changes in this content of mRNA or proteins in peripheral cells or visualizing dysregulated receptors in mind using advanced neuroimaging techniques, may be developed as useful transcriptional biomarkers..